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Chronic periodontitis, an inflammatory gum disease, influences prognosis and the severity of... PDF Print E-mail

Researchers from the University of Granada have demonstrated for the first time that chronic periodontitis, an inflammatory gum disease which provokes gradual teeth loss, is closely related to the severity of acute myocardial infarction, commonly known as heart attack.

In a pioneering research, published in the Journal of Dental Research, and titled "Acute myocardial infarct size is related to periodontitis extent and severity," this team has demonstrated that the extent and severity of chronic periodontitis is related to the size of acute myocardial infarction through seric levels of troponin I and myoglobin (biomarkers of myocardial necrosis).

This research results in part from the conclusions of Rafael Martín Marfil Álvarez's doctoral dissertation, which was directed by UGR professors Francisco Mesa Aguado (Stomatology Department), José Antonio Ramírez Hernández (Medicine Department), and Andrés Catena Martínez (Experimental Psychology Department). This research analysed 112 patients who had suffered from acute myocardial infarction, at the Virgen de las Nieves University Hospital Cardiology Unit. They all underwent a series of cardiological, biochemical and periodontal health checks and tests.

According to professor Francisco Aguado, one of the authors of this research (which will have to be confirmed through further research), "chronic periodontitis appears as a death risk factor and it plays an important role in the prognosis of acute myocardial infarction."

Researchers point out that it will be necessary to conduct follow-up checks with periodontal patients who have suffered myocardial infarction in order to determine the severity (or lack of it) of their clinical evolution (new coronary events, cardiac failure, or even death).

"If that happens to be the case, chronic periodontitis should be considered as a predictor in the development of myocardial infarction, and be therefore included in the risk stratification scores," according to Mesa Aguado.

Story Source:

The above story is based on materials provided by University of Granada. Note: Materials may be edited for content and length.

http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/dentistry/~3/VcPZqIPpt5c/150113090418.htm

 
A potential long-lasting treatment for sensitive teeth PDF Print E-mail

Rather than soothe and comfort, a hot cup of tea or cocoa can cause people with sensitive teeth a jolt of pain. But scientists are now developing a new biomaterial that can potentially rebuild worn enamel and reduce tooth sensitivity for an extended period. They describe the material, which they tested on dogs, in the journal ACS Nano.

Chun-Pin Lin and colleagues note that tooth sensitivity is one of the most common complaints among dental patients. Not only does it cause sharp pains, but it can also lead to more serious dental problems. The condition occurs when a tooth's enamel degrades, exposing tiny, porous tubes and allowing underlying nerves to become more vulnerable to hot and cold.

Current treatments, including special toothpastes, work by blocking the openings of the tubes. But the seal they create is superficial and doesn't stand up to the wear-and-tear of daily brushing and chewing. Lin's team wanted to find a more durable way to address the condition.

The researchers made a novel paste based on the elements found in teeth, namely calcium and phosphorus. They applied the mixture to dogs' teeth and found that it plugged exposed tubes more deeply than other treatments. This depth could be the key, the researchers conclude, to repairing damaged enamel and providing longer-lasting relief from tooth sensitivity.

Story Source:

The above story is based on materials provided by American Chemical Society. Note: Materials may be edited for content and length.

http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/dentistry/~3/_jTtxbN_KHc/150107123130.htm

 
Genome wide expression changes in vascular tissue identified due to infection/diet PDF Print E-mail

Although it has been shown that a diet high in fat and exposure to certain bacteria can cause atherosclerosis (the buildup of fats, cholesterol and other substances on artery walls which can restrict blood flow), researchers have for the first time identified distinct gene pathways that are altered by these different stimuli. These findings, which currently appear in BMC Genomics, suggest that future therapies for this disease may need to be individualized.

Atherosclerosis is a common human disease associated with heart attack and stroke. Certain bacteria as well as high fat diet are associated with an increased risk for atherosclerosis. One of these bacteria, Porphyromonas gingivalis, is found in the mouth of humans with periodontal disease; another, Chlamydia pneumoniae, causes pneumonia.

In this study, the researchers used four experimental groups to compare genome-wide expression changes in vascular tissue. The first group was subjected to Porphyromonas gingivalis while the second group received Chlamydia pneumoniae. The third group was placed on a high-fat diet while the fourth group was the control. In collaboration with the Clinical and Translational Science Institute (CTSI) at Boston University, the researchers performed genome-wide microarray profiling and analysis of vascular tissue from all groups to reveal gene pathways altered in the atherosclerotic plaque by each treatment group.

"Given the prevalence of diet-induced obesity and infection with Porphyromonas gingivalis and Chlamydia pneumoniae in the general population and the likelihood of co-morbidity of obesity with chronic or recurring infection with these common pathogens, these findings suggest that the development of atherosclerosis in humans is likely more complex and multifactorial than previously appreciated," explained senior author Caroline Attardo Genco, PhD, professor of medicine and microbiology at BUSM. "These findings may explain how specific infections or a high-fat diet may cause atherosclerotic plaques to undergo changes which affect their size and stability and may ultimately lead to a heart attack," she added.

Story Source:

The above story is based on materials provided by Boston University Medical Center. Note: Materials may be edited for content and length.

http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/dentistry/~3/GaCvTecChqw/150107101403.htm

 
Teeth Whitening Home Remedies: Simple & Effective Way to Get Whiter Teeth PDF Print E-mail

By Alex Stacanov

Teeth whitening home remedies are one of simple, effective and harmless way to adorn you with beautiful flawless white teeth that are pearly like white crystals. The importance of white teeth are often known to people who are in need of public support, who work among the people, socialists etc. for only when there is […]

The post Teeth Whitening Home Remedies: Simple & Effective Way to Get Whiter Teeth appeared first on Worldental.Org.

 
Six New Year Oral Care Resolutions PDF Print E-mail

By marvyb

With the onset of New Year, everyone makes certain resolutions. While some decide to overcome and control their bad habits, some make plans about achieving something, but have you ever considered making any resolution for the well being of your teeth and gums? Our health and body deserve the most attention and care than anything else and this […]

The post Six New Year Oral Care Resolutions appeared first on Worldental.Org.

 
Byproducts from bacteria awaken dormant T-cells, HIV viruses PDF Print E-mail

Dental and medical researchers from Case Western Reserve University found another reason to treat periodontal disease as soon as possible.

They discovered that byproducts of bacteria in gum disease, called metabolic small chain fatty acid (SCFA), can work together to wake up HIV in dormant T-cells and cause the virus to replicate.

Their findings help explain why people with the HIV -infections and periodontal disease have higher levels of the virus in their saliva than HIV patients with healthy gums.

The researchers speculate that byproducts from other bacteria infections in other diseases might change gene expression using similar mechanisms.

For dental patients with HIV, their findings further support how important it is to treat bacterial infections in gum disease early.

This interaction by SCFA and T-cells surprised co-investigators Fengchun Ye, assistant professor of biological sciences at the Case Western Reserve University School of Dental Medicine, and Jonathan Karn, director of the Center for Aids Research and professor and chair of the Department of Molecular Biology and Microbiology at Case Western Reserve's medical school.

Their findings are described in the article, "Short chain fatty acids potently induce latent HIV-1 in T-cells by activating P-TEFb and multiple histone modifications," published in January 2015 in the journal Virology.

In the interaction between gum disease and HIV, five SCFA byproducts from two prevalent oral bacteria -- Porphyromonas gingivalis (Pg) and Fusobacterium nucleatum (Fn) -- are involved in activating resting immune T-cells carrying latent (inactive) HIV-1 virus.

The process acts much like the jumper cables attached to a live battery recharging a dead one to get it running again, according the researchers.

Ye explained that all humans have a reservoir of resting T-cells that wake up and respond to inflammation to ward off an infection in the body.

"As long as someone is healthy, the reservoir remains untapped," he said.

But for people with HIV, these T-cells can also have the sleeper HIV-1 virus, which remains in a dormant state until awakened, Karn said.

Last year, Ye and Karn discovered that one SCFA -- butyric acid -- induced a chain of events that reactivate the virus associated with Kaposi's sarcoma, the most common malignancy in HIV patients.

Following that discovery, the researchers expanded their investigation to all SCFAs. They found that a high quantity of butyric acid activates the T-cell and incites virus replication. But smaller amounts of the five SCFA, working together, have the same impact.

"Looking at only butyric acid was misleading," said Karn, the Reinberger Professor of Molecular Biology. "It surprised us to find they all work as an aggregate."

The impact on waking up T-cells and activating HIV replication was a "double whammy" find that contributes to understanding the little-known microbiome in HIV disease, Karn said.

That prompted the researchers to investigate the mechanism that drives the replication of the virus in gum disease.

HIV antiviral therapy prevents active HIV cells from replicating and doesn't affect the quiet viruses in sleeping T-cells.

As long as the patient is free of gum disease, the virus sleeps and remains in check, Karn said.

Story Source:

The above story is based on materials provided by Case Western Reserve University. Note: Materials may be edited for content and length.

http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/dentistry/~3/xEgi1BYEUYY/150105112500.htm

 
Covington Who's Who Selects Dr. Robert Palmieri as a VIP Member of the Executive and... PDF Print E-mail
The selection recognizes Dr. Roberto Palmieri's commitment to excellence in Healthcare.
 
Latest Cone Beam CT and Dental Laser Tool Makes Surgery More Comfortable and Less Invasive PDF Print E-mail
Dr. Timothy Kosinski introduced intra-oral cancer screening with Velscope technology that is used to assist dentists in discovering cancerous or pre-cancerous growths in patients 18 and older that may not be apparent to the naked eye.
 
UPCD Centennial Lecture Series - I PDF Print E-mail
photos below (http://updent.blogspot.com/2014/12/upcd-centennial-lecture-series-i.html).How to pay:You can deposit payment thru:Bank of the Philippine Islands (BPI)Ground Floor Banko CenterOrtigas Avenue, Greenhills San...
 
UPCD Centennial Lecture Series - I PDF Print E-mail
Congratulations for a very successful event. The Centennial Levture held at the Lung Center last February 3...
 
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